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This hypothesis is widely accepted in the pathogenesis of PE 28

Progress of depression mechanism based on omics method

The catabolism of 2-HG is catalyzed by stereospecific dehydrogenases, D-2-hydroxyglutarate dehydrogenase and L-2-hydroxyglutarate dehydrogenase

Pharmacological targeting of CHAC1-mediated processes therefore represents a rational therapeutic strategy, potentially disrupting the cycle of GSH depletion, ER stress amplification and iron-dependent peroxidation that drives neuronal loss

The difference might be closely related to the special microenvironment of iron overload in ectopic endometrial stromal cells